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for example, the autophagy receptor hippocalcin-like 1 (HPCAL1) promotes ferroptosis yet paradoxically facilitates NSCLC growth in vivo, suggesting that its net oncogenic role may involve pleiotropic functions beyond regulating this cell death pathway (77)

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This triggers GH release through a separate signaling cascade

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Moreover, another ferroptosis-related lncRNA risk model demonstrated considerable clinical significance in predicting breast cancer prognosis and response to immunotherapy

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